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AB9311

Sigma-Aldrich

Anti-Vasopressin V1B Receptor. Antibody

Chemicon®, from rabbit

Synonym(s):

V1B Receptor

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

rabbit

Quality Level

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

purified by

affinity chromatography

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

immunohistochemistry: suitable (paraffin)

UniProt accession no.

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... AVPR1B(553)

Specificity

Other species have not been tested. The immunogen has 62% homology with mouse and 62% with rat.
Recognizes Vasopressin V1B Receptor.

Immunogen

Synthetic peptide from the 3rd cytoplasmic domain of human Vasopressin V1B Receptor.

Application

Immunohistochemistry on paraffin embedded tissue sections.
Optimal working dilutions must be determined by the end user.
Research Category
Neuroscience
Research Sub Category
Neurochemistry & Neurotrophins

Neuroinflammation & Pain
This Anti-Vasopressin V1B Receptor. Antibody is validated for use in IH(P) for the detection of Vasopressin V1B Receptor.

Physical form

Liquid in PBS, pH 7.7 with 0.01% sodium azide.

Storage and Stability

Maintain at -70°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


Certificates of Analysis (COA)

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S Hossein Fatemi et al.
Schizophrenia research, 112(1-3), 46-53 (2009-06-03)
Prenatal viral infection has been associated with the development of schizophrenia and autism. Our laboratory has previously shown that viral infection causes deleterious effects on brain structure and function in mouse offspring following late first trimester (E9) and late second
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Journal of neuropathology and experimental neurology, 71(1), 54-65 (2011-12-14)
The human polyomavirus JC (JCV) infects glial cells and causes progressive multifocal leukoencephalopathy (PML), a demyelinating disease of the brain, in immunosuppressed individuals. The extent of JCV infection of neurons is unclear. We determined the prevalence and pattern of JCV

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