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MAB2059Z

Sigma-Aldrich

Anti-Integrin β4 Antibody, clone ASC-8 (Azide Free)

clone ASC-8, Chemicon®, from mouse

Synonym(s):

CD104, MAB2059

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

Quality Level

100

antibody form

purified antibody

antibody product type

primary antibodies

clone

ASC-8, monoclonal

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

flow cytometry: suitable
immunohistochemistry: suitable
immunoprecipitation (IP): suitable

isotype

IgG1κ

NCBI accession no.

NM_000213.3
NM_001005619.1
NM_001005731.1

UniProt accession no.

P16144

shipped in

wet ice

target post-translational modification

unmodified

Gene Information

human ... ITGB4(3691)

Related Categories

Application

Anti-Integrin β4 Antibody, clone ASC-8 (Azide Free) is an antibody against Integrin β4 for use in FC, IP, IH, FUNC.

Quality

Flow Cytometry Analysis:
0.5 µg of the antibody was used to detect Integrin β4 in 1x10^6 A431 cells.
0.5 µg of the antibody was used to detect Integrin β4 in 1x10^6 HeLa cells.

Physical form

Format: Purified

Other Notes

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Integrin-blocking antibodies delay keratinocyte re-epithelialization in a human three-dimensional wound healing model.
Egles, Christophe, et al.
Testing, 5, e10528-e10528 (2010)
Matthias Rath et al.
Cellular and molecular life sciences : CMLS, 79(6), 340-340 (2022-06-07)
Cerebral cavernous malformations (CCM) are low-flow vascular lesions prone to cause severe hemorrhage-associated neurological complications. Pathogenic germline variants in CCM1, CCM2, or CCM3 can be identified in nearly 100% of CCM patients with a positive family history. In line with

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