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M1555

Sigma-Aldrich

MAHMA NONOate

Synonym(s):

6-(2-Hydroxy-1-methyl-2-nitrosohydrazino)-N-methyl-1-hexanamine, NOC-9

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About This Item

Empirical Formula (Hill Notation):
C8H20N4O2
CAS Number:
146724-86-9
Molecular Weight:
204.27
MDL number:
MFCD01310509
UNSPSC Code:
12352106
PubChem Substance ID:
24896668
NACRES:
NA.25

Quality Level

200

shipped in

dry ice

storage temp.

−70°C

SMILES string

[H][N+]([H])(C)CCCCCCN(C)N([O-])N=O

InChI

1S/C8H19N4O2/c1-9-7-5-3-4-6-8-11(2)12(14)10-13/h9H,3-8H2,1-2H3/q-1/p+1

InChI key

WASRCWNCCFPNEJ-UHFFFAOYSA-O

Application

NO donor

Biochem/physiol Actions

NO release: t1/2 = 3 min (phosphate buffer at 22° C)

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

Certificates of Analysis (COA)

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Philip E James et al.
Circulation research, 94(7), 976-983 (2004-02-14)
Vascular dysfunction in diabetes is attributed to lack of bioavailable nitric oxide (NO) and is postulated as a primary cause of small vessel complications as a result of poor glycemic control. Although it has been proposed that NO is bound
D Clark et al.
Molecular plant-microbe interactions : MPMI, 13(12), 1380-1384 (2000-12-06)
We used a variety of nitric oxide (NO) donors to demonstrate that NO inhibits the activities of tobacco catalase and ascorbate peroxidase (APX). This inhibition appears to be reversible because removal of the NO donor led to a significant recovery
Stefan Muenster et al.
Anesthesiology, 125(5), 952-963 (2016-10-19)
Transfusion of packed erythrocytes stored for a long duration is associated with increased pulmonary arterial pressure and vascular resistance. Prolonged storage decreases erythrocyte deformability, and older erythrocytes are rapidly removed from the circulation after transfusion. The authors studied whether treating
H P Monteiro et al.
Free radical biology & medicine, 28(2), 174-182 (2001-04-03)
Nitric oxide (NO) can participate in cellular signaling. In this study, monoclonal antibodies against proteins from the growth factor-mediated signalling pathway were used to identify a set of 126-, 56-, 43-, and 40-kDa proteins phosphorylated on tyrosine at NO stimulation
S H Lin et al.
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 20(9), 1380-1391 (2000-09-20)
Neuronal injury may be dependent upon the generation of the free radical nitric oxide (NO) and the subsequent induction of programmed cell death (PCD). Although the nature of this injury may be both preventable and reversible, the underlying mechanisms that
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