Skip to Content
MilliporeSigma
All Photos(2)

Documents

SAB5200003

Sigma-Aldrich

Monoclonal Anti-DLG4 antibody produced in mouse

clone 7E3, 1 mg/mL, purified immunoglobulin

Synonym(s):

Anti-DLG4, Anti-PSD95, Anti-SAP90

Sign Into View Organizational & Contract Pricing
All Photos(2)

About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

mouse

Quality Level

100

conjugate

unconjugated

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

7E3, monoclonal

form

buffered aqueous glycerol solution

mol wt

antigen predicted mol wt 100 kDa

species reactivity

mouse, rat, bovine

concentration

1 mg/mL

technique(s)

western blot: suitable

isotype

IgG2a

NCBI accession no.

NP_062567.1

UniProt accession no.

Q62108

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

rat ... Dlg4(29495)

Specificity

Detects a ~100 kDa protein corresponding to the molecular mass of PSD-95 on SDS PAGE immunoblots. Additional cross-reactive bands are detected at ~80 kDa and ~50 kDa in rat and mouse samples.

Immunogen

Recombinant rat PSD-95, Native Sequence

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Solution in PBS, pH 7.4, 50% glycerol, and 0.09% sodium azide

Not finding the right product?  

Try our Product Selector Tool.

wgk_germany

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

Already Own This Product?

Find documentation for the products that you have recently purchased in the Document Library.

Visit the Document Library

Xiao-Yan Zhang et al.
The Journal of biological chemistry, 289(45), 31200-31211 (2014-09-19)
Glycine can persistently potentiate or depress AMPA responses through differential actions on two binding sites: NMDA and glycine receptors. Whether glycine can induce long-lasting modifications in NMDA responses, however, remains unknown. Here, we report that glycine induces long-term potentiation (LTP)
Monika Vrajová et al.
Neurochemical research, 39(11), 2040-2046 (2014-08-01)
There is accumulating evidence that methamphetamine (MA) is a widely abused drug popular among pregnant women. MA exposure is associated with changes in the function of neurotransmitter systems, namely the dopaminergic, serotonergic and glutamatergic systems. Since N-methyl-D-aspartate receptors (NMDA) are
Soroush G Sadeghi et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 34(44), 14536-14550 (2014-10-31)
In the vestibular periphery a unique postsynaptic terminal, the calyx, completely covers the basolateral walls of type I hair cells and receives input from multiple ribbon synapses. To date, the functional role of this specialized synapse remains elusive. There is
Sven Akkerman et al.
PloS one, 9(11), e111692-e111692 (2014-11-06)
Drug effects are usually evaluated in animals housed under maximally standardized conditions. However, it is assumed that an enriched environment (EE) more closely resembles human conditions as compared to maximally standardized laboratory conditions. In the present study, we examined the
Huiling Qu et al.
Restorative neurology and neuroscience, 32(5), 597-609 (2014-07-19)
The mechanism underlying forced limb-use -induced structural plasticity remains to be studied. We examined whether the cyclic adenosine monophosphate (cAMP)-mediated signal transduction pathway was involved in brain plasticity and promoted behavioral recovery induced by forced limb-use after stroke. Adult rats
View All Related Papers

Our team of scientists has experience in all areas of research including Life Science, Material Science, Chemical Synthesis, Chromatography, Analytical and many others.

Contact Technical Service